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: You should learn to spell. Your credibility is nill when you can't spell. : : I recently found this information on the web and didn't know what to think of it. So I thought i'd let you (and others who visit this site) take a look at it and tell me what you think about it. Im still debating on whether to believe it or not. : : K-17 shares a number of traits with HIV. Both are retroviruses and : : both : : are extremely vulnerable to changes in blood pH and environment. : : HIV incubates in the host for an exceptionally long time, a necessity : : since : : it will eventually kill the host and the vectors are not common : : enough : : to ensure its spread in a shorter time. : : K-17 operates in a different manner. Almost as soon as infection : : occurs K-17 spreads throughout the host body, having an immediate : : effect. : : Various parts of the body are attacked and between 20 and 30 per : : cent of : : the virus DNA is transcribed into the host’s cells. About 90% of : : this : : transcription codes for intron structures, the rest being spread out : : evenly : : along the helix. : : The symptoms of K-17 infection are not always the same. If a : : host is : : not compatible (there is speculation that most Asian and Afro-Caribbean : : peoples are not susceptible - see below) then the virus will either : : survive : : in the bloodstream in diminished numbers (while still being : : contagious) : : or disappear completely, attacked by the body’s immune : : system. In : : compatible hosts the spread of the disease is more uniform. : : Within the first four days of infection the virus will breed and : : spread within the host, leaving RNA-based infectious viruses in almost : : every : : bodily fluid.. The individual effects are most easily dealt with : : by : : category: : : The Body: : : K-17 exhibits traits unlike any other known virus once : : established. : : The most dramatic effect is on the immune system. : : The virus causes increased activity from white cells, increasing : : their ‘kill’ sensitivity. This causes some parts of the hosts own body : : to : : suffer temporary attack. During this period the host will suffer : : vomiting : : and, occasionally, violent fits. : : At this point K-17 mutates to : : form a second, benign, virus. Cells in : : the bone marrow are attacked and : : are caused to inject a virus composed : : of : : a complete copy of the host’s DNA inside a modified K-17 protein shell. : : It is this action which finally silences the host’s immune system : : attack : : on the virus since K-17 type protein shells are now identified as : : belonging : : to the host. : : This second virus is extremely infectious, but with a slight : : difference. : : The changes in the K-17 protein shell make it impossible for : : this : : virus to survive outside of the host body, so it cannot be passed : : to : : another organism. It is also not always fatal to infected cells. Normal : : viral : : function occurs (virus attacks host cell. Virus penetrates host cell : : and : : injects its own DNA inside the cell wall. Protein substitution forces : : the host cell to burst, spraying out multiple copies of the original : : virus), : : but the protein substitution does not take place. Thus the host : : cells are : : re-infected with their own DNA. About one infection in three : : hundred results : : in the manufacture of new viral cells, keeping a roughly : : constant level : : of infection in the bloodstream. : : This process results in the host’s DNA being set in stone at the : : point of infection. Ageing has been linked to errors in DNA : : transcription : : when new cells are created. In a host infected with K-17 : : these errors : : will : : be ‘repaired’ by the benign virus. It seems likely that any longevity : : effects stem from this process. : : Negative effects from the restructuring of the immune system include : : the : : occasional mis-identification of the host’s own cells. This is most : : likely : : to occur when skin cells contain melanin from exposure to : : ultra-violet : : light (this may be an explanation to the viruses : : non-contagion to : : Afro-Caribbean races. A non-selective strain would kill : : an : : Afro-Caribbean : : host, along with itself, in a couple of days. This would : : allow a strain : : which would only use Afro-Caribbean people as carriers to : : evolve as the : : more successful organism). The slight modification of cell : : chemistry is : : enough for the newly paranoid immune system to kill them. : : Sufficient exposure to sunlight would cause destruction of the host’s : : skin : : by their own immune system. The skin would die and slough off , : : resulting : : in massive blood loss, blood poisoning by non-sterile contact : : and, rapidly, : : death. : : The Brain: : : This is where some of the most dramatic changes take place. : : The introduction of K-17 DNA in to the host makeup produces a : : number : : of effects. : : Blood flow to various areas of the brain is increased : : and : : neurotransmitter production is stimulated. : : The areas most affected are the Amyglada and the Visual Cortex (the : : areas : : around the brain stem and outer-back of the brain). Acetocoline : : production : : is stimulated and a localised attack on brain-cell proteins : : on : : the cortex is stimulated. : : The Amyglada is the most primitive part of the brain, linked mainly : : with : : the powerful fight-or-flight reflex. The host will suffer increased : : aggression : : and reactions, particularly to physically threatening : : situations. Arousal, : : either combative or sexual, will have a greater : : effect. It : : can also cause personality changes and mood swings since overriding : : the : : more powerful instincts become harder. : : The changes to the Visual Cortex cause vision to become more acute. : : Processing : : of images proceeds faster. Night vision is enhanced within : : certain parameters. : : The eyes themselves undergo no change, but a large : : part : : of human low light sight deficiency is due to the time it takes the : : brain to process darkened images (this has been exploited to produce 3D : : effects on TV with only the use of glasses with one darkened lens. As : : long : : as the camera moves in the right direction processing of the image : : from : : the shaded eye takes a fraction longer and the forebrain it presented : : with two images out of step with one another - the objects seen from two : : different viewpoints, hence 3D). : : The information collected by the eyes is unchanged, but it is : : processed : : better. : : The attack on cells on the cortex causes the formation of an engram on : : the upper cortex stretching across both sides of the brain. This attack : : is a side-effect of the modifications to the host’s immune systems. The : : result is a paraphilic attraction to blood (the instance of paraphilia : : has been linked to changes in the brain. Well documented cases include a : : man who became aroused by paper-clips while suffering from a brain : : tumour. : : Removal of the tumour ended this.). This attraction is entirely : : sexual : : and is not linked to any physical craving in the host, though the : : anaemia : : caused by immune system restructuring has led to : : misunderstandings. : : The formation of this engram may also cause other paraphilia : : which have : : been linked to brain structure dysfunction (Necrophilia has : : been : : suggested as a common side-effect due to the Vampire/Graveyard legends : : - : : there : : is no evidence for this. What is more likely is that formation of : : the : : engram could cause any number of minor or major paraphilic attractions : : and fetishes, depending on its severity). : : Looser Rumours: : : Some people believe that the virus also stimulates the Pineal : : gland : : as well as producers of other neurotransmitters. They have suggested : : that : : this could lead to increased psychic abilities. I, personally, discount : : this. I have seen no suggestion in anything I have read or stolen that : : K-17 affects the Pineal gland. Besides, the connection between the : : Pineal : : gland and psychic ability is an assumed one carried over from the : : eighteenth : : century when it was found to be in a position similar to that : : of : : the supposed ‘third eye’. : : It has been suggested that K-17 stimulates the Adrenal gland, : : giving : : increased strength. Again, there is no evidence for this. If the : : Adrenal : : was constantly stimulated the host would be constantly edgy and : : hyper. : : Stimulation of the adrenal glad takes place more readily in hosts : : due to : : the stimulation of the Amyglada, but nothing even begins to back : : up a : : case for constant stimulation, even assuming the human body could : : stand : : the punishment. : : I have heard that the military are researching K-17. Personally, : : I : : doubt this. : : Other: : : RV K-17 was first isolated in South Africa by the University of : : Cape : : Town in 1962, found by accident in a medical study into viral : : gastroenteritis. : : The large scale testing revealed a small number of : : black subjects : : infected with a benign virus which had not been seen before. : : Since all : : of the subjects came from the slum townships it was written off : : as a : : degenerative hantavirus spread by rodents. Research did not continue. : : It : : was named virus K-17 and forgotten about. : : By sometime around 1985 K-17 was being researched by the Glaxo : : Wellcome corporation (to name but one). Now known as RV K-17 research : : had : : begun when the records from the 1962 gastroenteritis studies were : : reviewed, : : revealing the existence of an apparently benign contagious virus. : : It was : : hoped that research into the function of benign retroviruses could : : give : : some clue pointing towards a cure for AIDS. : : By 1988 most information regarding K-17 was removed from the : : public : : domain. The University of Cape Town no longer makes the results of the 1962 study available, citing it as out of date information and directing the inquirer to the later studies done in the US. : : Current official status of RV K-17 is still that it is a mildly contagious benign strain of degenerate hantavirus, originating in South Africa.

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